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Tuesday, January 31, 2012
Will Listening to Mozart Really Make You Smarter? [Science]
True Blood Season 5 Promo Teases the Return Of...
HBO has not yet announced a season five premiere date for True Blood. But the network did release the first official trailer for the suspenseful, sex-based summer smash and it teases a rather major return.
Of whom? We won't give any hints for those who wish to remain surprised, but any loyal fans of the series will have a very clear idea after viewing:
In other major True Blood news, former Law & Order: SVU star Christopher Meloni has signed on for a significant role, while the season five premiere title has been revealed.
What will it be? That information and a lot more can be found in the True Blood spoilers section at TV Fanatic!
Source: http://www.thehollywoodgossip.com/2012/01/true-blood-season-5-promo-teases-the-return-of/
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Monday, January 30, 2012
Solar storm
Solar flare sets off auroras around the Arctic Circle
Web edition : 5:03 pm
Earth?s nearest star erupted late on January 22, belching out a solar flare. At the same time the sun launched a blast of hot gas known as a coronal mass ejection toward Earth.
A day later, when the particles slammed into the atmosphere, they set off auroras around the Arctic Circle. Fast-moving protons that came along for the ride also triggered a solar radiation storm, the biggest seen since the ?Halloween storms? of October 2003.
Scientists expect such powerful solar blasts to become more common as the sun moves toward a predicted peak of activity in 2013.
Found in: Astronomy and Atom & Cosmos
Source: http://www.sciencenews.org/view/generic/id/338026/title/Solar_storm
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Sunday, January 29, 2012
Obama urges Congress to act in election year (AP)
CAMBRIDGE, Md. ? President Barack Obama rallied House Democrats for an election-year fight, urging them to work with Republicans if they show some willingness to put politics aside but telling the rank and file to call them out if they stand in the way.
Addressing Democrats on the final day of their three-day annual retreat, Obama outlined the political stakes over the next few months as congressional Democrats try to push his agenda in the face of Republican opposition, the GOP choses its nominee and signs of recovery in a fragile economy go a long way to determining his re-election chances and the party's fate.
Obama said Democrats should seize the opportunity "whenever there is a possibility that the other side is putting some politics aside for just a nanosecond in order to get something done for the American people, we've got to be right there ready to meet them," the president told the sometimes raucous crowd.
However, "where they obstruct, where they're unwilling to act, where they're more interested in party than they are in country, more interested in the next election than the next generation, then we've got to call them out on it," the president said. "We've got to push. We can't wait; we can't be held back."
Coming off a three-day tour to promote his State of the Union message, Obama promised a "robust debate about whose vision is more promising" when Republicans choose their nominee.
On a day when reports showed the economy picking up late in 2011 but still considered "fragile" by the White House, Obama told Democrats wondering about their re-election prospects: "It's going to be a tough election because a lot of people are still hurting out there and a lot of people have lost faith generally about the capacity of Washington to get anything done."
House Republicans, who held their retreat in Baltimore last week, have repeatedly said the election will be a referendum on Obama's policies, especially his handling of the economy.
The president acknowledged that Democrats have embraced parts of his agenda when it was politically difficult and in some cases costly. The party took a drubbing in the midterm elections, losing control of the House and seeing their ranks diminished in the Senate.
And despite some past clashes with House Democrats over his willingness to compromise with Republicans, Obama was warmly received and was introduced as "our champion" by Rep. John Larson of Connecticut.
The president returned the warmth with a vote of confidence that Democrats would win back the House in November, making a nod to their leader as "soon-to-be once-again Speaker of the House Nancy Pelosi."
"I believe in you guys. You guys have had my back through some very tough times," said the president, who received a small gift ? a DVD of House Democrats singing Rev. Al Green's "Let's Stay Together."
Last week, at a fundraiser at the Apollo Theater in New York, Obama stood on the stage and crooned a line from the Green classic.
Democrats were upbeat at their three-day session, energized by Obama's State of the Union address and its populist themes as well as recent polls showing more Americans say the country is on the right track and approve of Obama's handling of the economy. Divisions in the Republican ranks that were on full display last year in the fight over extending the payroll tax cut and the bitter battle between Mitt Romney and Newt Gingrich for the GOP presidential nomination also lifted Democratic spirits.
But the relationship with the White House hasn't always been cordial. Vice President Joe Biden, who addressed the Democrats prior to Obama's speech, described some of the rough patches.
He noted that several members in the room were mad at him in December 2010 after Obama negotiated an extension of President George W. Bush's tax cuts over the objections of some House Democrats. Last year, frustrated Democrats complained the Obama gave away too much in negotiating a spending bill and an agreement to raise the government's borrowing authority.
Biden said Pelosi told him at the last conference to "get tough. Enough is enough." He said the "message was heard. The message was heard. And I think we've delivered."
Biden said Democrats would reclaim the House and he would help candidates in Pennsylvania, Ohio, Michigan, Iowa, New Hampshire and Florida. Those states also are critical to Obama's hopes of winning another term.
"We cannot succeed unless you all come back," Biden told House Democrats.
The vice president was more pointed in his political remarks than Obama and called out some Republicans by name. He said the American people will reject GOP unwillingness to compromise and its blatant determination to make Obama a one-term president.
Of the presidential candidates, Biden said Romney's criticism of the auto bailout and a host of positions stated by rival Newt Gingrich on government intervention will create a clear contrast for voters.
"These guys are helping us by saying what they believe," Biden said.
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UFC on Fox 2 picks, Vegas-style: Bisping really getting annoyed by Sonnen?s wrestling lines
CHICAGO -- It's all about staying off your back and top control in tonight's three big fights on the UFC on Fox 2 card.
If they control the pace in fights, top level wrestlers have the ability to steal rounds and money. A mix of underdogs and favorites (Chael Sonnen, Phil Davis and Chris Weidman) with excellent college wrestling backgrounds, will be an interesting follow on the betting scene.
Sonnen's controlled the pace so well in his recent fights, it's made him just about unbeatable and a huge favorite over Michael Bisping. We'll find out tonight if Bisping's takedown defense allows him to stay in the fight. Hopefully keeps his cool. Sonnen along with yesterday's weigh-in crowd at the Chicago Theater really got under the Brit's skin.
As an underdog, Phil Davis will be looking to do slow things down against Rashad Evans. Chris Weidman's in the most interesting position because the former Hofstra star could probably stay on top of Demian Maia the entire fight, but that's incredibly dangerous place to be against a top level jiu-jitsu fighter. In spite of that Weidman is a pretty solid favorite over the UFC veteran.
Former UFC bantamweight champ Miguel Torres joined Yahoo! Sports' Kevin Iole and myself on ESPN1100/98.9 FM to pick the fights using the Las Vegas' numbers.
UFC on Fox 2 odds:
Best plays in bold
Rashad Evans (-205) vs. Phil Davis (+175) - Light heavyweight
Michael Bisping (+360) vs. Chael Sonnen (-450) - Middleweight
Demian Maia (+120) vs. Chris Weidman (-140) - Middleweight
Evan Dunham (-355) vs. Nik Lentz (+295) - Lightweight
Mike Russow (-150) VS. Jon-Olav Einemo (+130) - Heavyweight
George Roop (-115) vs. Cub Swanson (-105) - Featherweight
Charles Oliveira (-490) vs. Eric Wisely (+390) - Featherweight
Shane Roller (-225) vs. Michael Johnson (+185) - Lightweight
Joey Beltran (-205) vs. Lavar Johnson (+175) - Heavyweight
Chris Camozzi (-150) vs. Dustin Jacoby (+130) - Middleweight
Saturday, January 28, 2012
ABC picks up 3 comedy pilots (omg!)
LOS ANGELES, Jan 27 (TheWrap.com) - ABC beefed up its comedy stockpile Friday with orders for three pilots. "How to Live With Your Parents for the Rest of Your Life," created by Claudia Lonow ("Friends With Benefits," "Accidentally on Purpose" and, more recently, the troubled cross-dressing sitcom "Work It"), will follow the adventures of recent divorcee and single mother Polly, who moves in with her eccentric parents, Elaine and Max, who are full of life but know no boundaries. Lonow is also executive-producing the half-hour, single-camera project, which comes via 20th Century Fox and Imagine Television. Brian Grazer and Francie Calfo are also attached as executive producers.
The network has also given the go-ahead to a pilot by Adam Sztykiel, writer of the big-screen offerings "Due Date" and "Made of Honor." The as-yet-untitled project promises to offer a "raw, hilarious peek behind the curtain of modern 20-something relationships." Sztykiel created and will co-executive produce the project through 20th Century Fox Television. Sean Perrone and Aaron Kaplan ("Made of Honor," "You, Me and Dupree") are executive-producing the half-hour, single-camera comedy.
Rounding out the trio is "Only Fools and Horses," which is based on the British series of the same name and "chronicles the misadventures of two streetwise brothers and their aging grandfather as they concoct outrageous, morally questionable get rich quick schemes in their quest to become millionaires."
Steven Cragg and Brian Bradley, both veterans of "MADtv," are writing the half-hour, multi-camera project, which comes via ABC Studios.
(Editing By Zorianna Kit)
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Broncos hire Jack Del Rio as defensive coordinator (AP)
DENVER ? John Fox and Jack Del Rio are together again.
The Denver Broncos announced Friday night they had agreed to terms with Del Rio to become the club's new defensive coordinator.
Del Rio was Fox's first defensive coordinator in Carolina in 2002 before leaving after one year to coach the Jacksonville Jaguars, who fired him in November.
Del Rio replaces Dennis Allen, who left after one year in Denver to coach the Oakland Raiders.
Del Rio is the Broncos' seventh defensive coordinator in seven seasons. Other men who have filled the Mile High musical chair in the last six seasons are Larry Coyer (2006), Jim Bates (2007), Bob Slowik (2008), Mike Nolan (2009) and Don Martindale (2010). Allen was the only one who left for a head coaching job.
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Friday, January 27, 2012
Mutated Kras spins a molecular loop that launches pancreatic cancer
![[ Back to EurekAlert! ]](http://www.eurekalert.org/images/back2e.gif){kind=small}
Public release date: 26-Jan-2012[ | E-mail |
Share ] Contact: Scott Merville
smerville@mdanderson.org
713-792-0661
University of Texas M. D. Anderson Cancer Center
UT MD Anderson-led team identifies new potential treatment avenue to block an elusive target
HOUSTON Scientists have connected two signature characteristics of pancreatic cancer, identifying a self-perpetuating "vicious cycle" of molecular activity and a new potential target for drugs to treat one of the most lehal forms of cancer.
The research, reported in the journal Cancer Cell and led by scientists at The University of Texas MD Anderson Cancer Center, connected the molecular dots between:
- Mutated versions of Kras, a gene that acts as a molecular on-off switch but gets stuck in the "on" position when mutated.
- Heightened activity of a protein complex called NF-?B that controls activation of genes.
"Kras is mutated in 80 to 95 percent of pancreatic ductal adenocarcinomas, and is the most frequent mutation among all cancers," said senior author Paul Chiao, Ph.D., professor in MD Anderson's Department of Molecular and Cellular Oncology.
About 42,000 new cases of pancreatic ductal adenocarcinoma are diagnosed in the United States each year. Estimates vary, but the 5-year survival rate has been 1 to 3 percent for decades and median survival after diagnosis is six months, the researchers note.
Interleukin-1? is a new potential drug target
"There have been many attempts to inhibit mutated Kras, but it's an elusive target that so far has defied treatment," Chiao said. "So if we can't hit Kras, maybe we can target one of its downstream genes. This research identifies some of those genes and suggests that interleukin-1apha (IL-1?) is a potential therapeutic target."
Chiao and colleagues identified IL-1? as a crucial player in a feed-forward loop that:
- Begins with mutationally activated Kras triggering a chain reaction that induces IL-1? expression;
- This in turn activates NF-?B via the protein kinase IKK2/?, which blocks the inhibitor of NF-?B.
- In the cell nucleus, NF-?B oversees gene transcription and regulates a number of inflammation-promoting genes, including IL-1?.
- IL-1? and another protein called p62 activate NF-?B which in turn cycles back to perpetuate the loop by activating its activators.
"It's a vicious cycle," Chiao said. The overactive NF-?B fuels pancreatic cancer by activating genes that promote inflammation, the growth of new blood vessels and block programmed cell death.
Chiao has three research grants from the National Cancer Institute to study pancreatic cancer. "We study signaling transduction pathways to try to find out why it's such a bad disease and to find a weak point for targeted therapy," he said.
In the Cancer Cell paper, the authors conclude: "Our findings suggest that the prime mover responsible for cancer-related inflammatory response and the development of pancreatic intraepithelial neoplasia (precancerous lesions) and pancreatic ductal adenocarcinoma is the mutant Kras-initiated constitutive activation of NF-?B."
This process, they further noted, creates a pro-tumor microenvironment by promoting inflammation, creation of new blood vessels and tissue repair that is similar to conditions found in inherited pancreatitis, inflammation of the pancreas that is linked to the development of cancer.
Kras mutation, IL-1?, NF-?B go together with poor survival
The team analyzed mouse and human tumors and mouse strains with mutated Kras expressed in their pancreases. In a series of experiments they found:
- Active IKK2/beta the activator of NF-?B was required for the Kras-mutated mice to develop either pancreatic cancer or precancerous legions.
- Deletion of IKK2/Beta interrupted Kras-stimulated inflammation and cell proliferation, suggesting that chronic inflammation is a key factor in promoting pancreatic cancer development.
- Microarray profiles of gene expression showed that several NF-?B-regulated inflammatory genes were present in high levels in mice with mutated Kras and active IKK2/beta but only found at lower levels in mice with IKK2/beta knocked out.
- In human pancreatic tumors, high expression of the same inflammatory genes in the mutated Kras mice were associated with positive lymph node status, high-risk, late tumor stage and poor survival.
- Expression of several genes regulated by NF-?B progressed from low levels in normal pancreases to higher levels in precancerous lesions and tumors, including IL-?.
- IL-1? was known to be both a target of and an inducer of NF-?B, but its expression had not previously been connected to mutated Kras. The team found that downstream targets of Kras, including IL-1?, are interrupted when IKK2/? is inactivated.
- Analysis of 14 human pancreatic cancer tumor samples showed that overexpression of IL-1?, the presence of Kras mutation and the activation of NF-?B are correlated and are associated with poor survival.
- Continued activation of NF-?B and its gene transcription activity are sustained by IL-1? and p62.
###
Co-authors with Chiao are Jianhua Ling, Ph.D., Rulying Zhao, M.D., Ph.D., Qianghua Xia, Ph.D., Zhe Chang, Ph.D., and Mien-Chie Hung, Ph.D., of MD Anderson's Department of Molecular and Cellular Oncology; Ya'an Kang, M.D., Ph.D., and Jason Fleming, M.D., of MD Anderson's Department of Surgical Oncology; Huamin Wang, M.D., Ph.D., and Jinsong Liu, M.D., Ph.D., of MD Anderson's Department of Pathology; Dung-Fang Lee, Ph.D., and Ihor Lemischka, Ph.D., of the Black Family Stem Cell Institute of Mount Sinai School of Medicine; Jin Li, Ph.D., of the Center for Applied Genomics of the Children's Hospital of Philadelphia; and Bailu Peng, Ph.D. of the Guangdong Entomological Institute, Guangdong, China.
The team's research was funded by grants from the National Cancer Institute, including MD Anderson's Cancer Center Core Support Grant.
[ | E-mail | Share ] ?
AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.
Public release date: 26-Jan-2012[ | E-mail |
Share ] Contact: Scott Merville
smerville@mdanderson.org
713-792-0661
University of Texas M. D. Anderson Cancer Center
UT MD Anderson-led team identifies new potential treatment avenue to block an elusive target
HOUSTON Scientists have connected two signature characteristics of pancreatic cancer, identifying a self-perpetuating "vicious cycle" of molecular activity and a new potential target for drugs to treat one of the most lehal forms of cancer.
The research, reported in the journal Cancer Cell and led by scientists at The University of Texas MD Anderson Cancer Center, connected the molecular dots between:
- Mutated versions of Kras, a gene that acts as a molecular on-off switch but gets stuck in the "on" position when mutated.
- Heightened activity of a protein complex called NF-?B that controls activation of genes.
"Kras is mutated in 80 to 95 percent of pancreatic ductal adenocarcinomas, and is the most frequent mutation among all cancers," said senior author Paul Chiao, Ph.D., professor in MD Anderson's Department of Molecular and Cellular Oncology.
About 42,000 new cases of pancreatic ductal adenocarcinoma are diagnosed in the United States each year. Estimates vary, but the 5-year survival rate has been 1 to 3 percent for decades and median survival after diagnosis is six months, the researchers note.
Interleukin-1? is a new potential drug target
"There have been many attempts to inhibit mutated Kras, but it's an elusive target that so far has defied treatment," Chiao said. "So if we can't hit Kras, maybe we can target one of its downstream genes. This research identifies some of those genes and suggests that interleukin-1apha (IL-1?) is a potential therapeutic target."
Chiao and colleagues identified IL-1? as a crucial player in a feed-forward loop that:
- Begins with mutationally activated Kras triggering a chain reaction that induces IL-1? expression;
- This in turn activates NF-?B via the protein kinase IKK2/?, which blocks the inhibitor of NF-?B.
- In the cell nucleus, NF-?B oversees gene transcription and regulates a number of inflammation-promoting genes, including IL-1?.
- IL-1? and another protein called p62 activate NF-?B which in turn cycles back to perpetuate the loop by activating its activators.
"It's a vicious cycle," Chiao said. The overactive NF-?B fuels pancreatic cancer by activating genes that promote inflammation, the growth of new blood vessels and block programmed cell death.
Chiao has three research grants from the National Cancer Institute to study pancreatic cancer. "We study signaling transduction pathways to try to find out why it's such a bad disease and to find a weak point for targeted therapy," he said.
In the Cancer Cell paper, the authors conclude: "Our findings suggest that the prime mover responsible for cancer-related inflammatory response and the development of pancreatic intraepithelial neoplasia (precancerous lesions) and pancreatic ductal adenocarcinoma is the mutant Kras-initiated constitutive activation of NF-?B."
This process, they further noted, creates a pro-tumor microenvironment by promoting inflammation, creation of new blood vessels and tissue repair that is similar to conditions found in inherited pancreatitis, inflammation of the pancreas that is linked to the development of cancer.
Kras mutation, IL-1?, NF-?B go together with poor survival
The team analyzed mouse and human tumors and mouse strains with mutated Kras expressed in their pancreases. In a series of experiments they found:
- Active IKK2/beta the activator of NF-?B was required for the Kras-mutated mice to develop either pancreatic cancer or precancerous legions.
- Deletion of IKK2/Beta interrupted Kras-stimulated inflammation and cell proliferation, suggesting that chronic inflammation is a key factor in promoting pancreatic cancer development.
- Microarray profiles of gene expression showed that several NF-?B-regulated inflammatory genes were present in high levels in mice with mutated Kras and active IKK2/beta but only found at lower levels in mice with IKK2/beta knocked out.
- In human pancreatic tumors, high expression of the same inflammatory genes in the mutated Kras mice were associated with positive lymph node status, high-risk, late tumor stage and poor survival.
- Expression of several genes regulated by NF-?B progressed from low levels in normal pancreases to higher levels in precancerous lesions and tumors, including IL-?.
- IL-1? was known to be both a target of and an inducer of NF-?B, but its expression had not previously been connected to mutated Kras. The team found that downstream targets of Kras, including IL-1?, are interrupted when IKK2/? is inactivated.
- Analysis of 14 human pancreatic cancer tumor samples showed that overexpression of IL-1?, the presence of Kras mutation and the activation of NF-?B are correlated and are associated with poor survival.
- Continued activation of NF-?B and its gene transcription activity are sustained by IL-1? and p62.
###
Co-authors with Chiao are Jianhua Ling, Ph.D., Rulying Zhao, M.D., Ph.D., Qianghua Xia, Ph.D., Zhe Chang, Ph.D., and Mien-Chie Hung, Ph.D., of MD Anderson's Department of Molecular and Cellular Oncology; Ya'an Kang, M.D., Ph.D., and Jason Fleming, M.D., of MD Anderson's Department of Surgical Oncology; Huamin Wang, M.D., Ph.D., and Jinsong Liu, M.D., Ph.D., of MD Anderson's Department of Pathology; Dung-Fang Lee, Ph.D., and Ihor Lemischka, Ph.D., of the Black Family Stem Cell Institute of Mount Sinai School of Medicine; Jin Li, Ph.D., of the Center for Applied Genomics of the Children's Hospital of Philadelphia; and Bailu Peng, Ph.D. of the Guangdong Entomological Institute, Guangdong, China.
The team's research was funded by grants from the National Cancer Institute, including MD Anderson's Cancer Center Core Support Grant.
[ | E-mail | Share ] ?
AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.
Source: http://www.eurekalert.org/pub_releases/2012-01/uotm-mks012612.php
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Utah teens arrested in alleged school bombing plot (AP)
SALT LAKE CITY ? A Utah high school student bragged to police that he was smarter than the Columbine killers and was plotting with an older student to set off a bomb during a school assembly and escape in a stolen plane, court documents say.
Dallin Morgan, 18, and the 16-year-old boy were pulled out of school Wednesday and arrested after authorities learned of the plot, Roy police spokeswoman Anna Bond said Thursday.
The students prepared by logging hundreds of hours on flight simulator software on their home computers, and they planned to take a plane at Ogden Hinckley Airport after the bombing, Bond said.
The juvenile hinted at the plan in text messages to a friend, writing that both suspects wanted "revenge on the world" and "we have a plan to get away with it too."
He hinted at the plan by writing "explosives, airport, airplane" and added, "We're just gonna kill and fly our way to a country that won't send us back to the US," according to a probable cause statement police filed to make the arrests late Wednesday.
The Associated Press isn't naming the 16-year-old because he is a minor.
The juvenile told investigators he was so "fascinated" by the 1999 Columbine High School massacre that he visited the Littleton, Colo., school and interviewed the principal about the shootings that killed 13 people. Roy police said the principal, Frank DeAngelis, confirmed that the boy made his visit Dec. 12.
Morgan was being held on $10,000 bail at Weber County jail on suspicion of conspiracy to commit mass destruction. The juvenile was in custody at Weber Valley Detention Center on the same charge. Prosecutors were weighing possible additional charges.
Both students had "absolute knowledge of the security systems and the layout of the school," Bond said. "They knew where the security cameras were. Their original plan was to set off explosives during an assembly. We don't know what date they were planning to do this, but they had been planning it for months."
School officials said there were no imminent plans to hold a school assembly.
Local and federal agents searched the school, two vehicles belonging to the suspects and their homes but found no explosives. The FBI is examining the suspects' computers, police said.
The parents of both students "woke up in the middle of a nightmare," Bond said. "They've been very cooperative."
The other Roy High School student who received text messages tipped authorities to the plot Wednesday, said the school's safety specialist, Nate Taggart.
The student "came forward and had some suspicions but not a lot of information ? enough that it gave administration the ability to make some connections and identify the students involved," Taggart said.
The school has about 1,500 students.
___
Associated Press writer Michelle Rindels in Las Vegas contributed to this report.
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Thursday, January 26, 2012
Analysis: More, not less, oil this year despite Iran ban (Reuters)
LONDON (Reuters) ? The world is likely to have more oil, not less, this summer even as Europe imposes sanctions on Iran over its nuclear program.
Although Europe's refiners will have to pay up for other sources of oil, they should have little difficulty finding them.
Extra crude oil from Saudi Arabia, Iraq and Libya will more than make up for any lost from Iran after the ban is imposed on July 1, and this is likely to be reflected in oil prices.
As much as 1 million barrels per day (bpd) more crude oil could be coming from these three producers alone - perhaps double the volume of Iranian exports lost to the European Union.
"The oil market should be very well supplied this summer - even better than now," said Samuel Ciszuk, Middle East and North African (MENA) analyst at consultancy KBC Energy Economics.
"Volumes from Iraq should be up significantly, Libya is doing very well and Saudi Arabia will increase production to compensate for some of the lost Iranian barrels."
The International Monetary Fund said on Wednesday sanctions against Tehran would imply supply declines of about 1.5 million bpd from the world's fifth largest oil producer, adding global oil prices could rise as much as 30 percent if Iran halted oil exports as a result of the West's actions.
But senior oil executives, traders and strategists see little chance of significant supply disruption this summer.
Although oil prices have risen on fears that conflict between Iran and the West could disrupt exports from the Middle East, they argue such a clash is extremely unlikely.
And with the European economy in the doldrums and Asian growth slowing, overall oil demand growth is being constrained.
"NOTHING WILL CHANGE"
Iran will keep selling its oil, much of it into Asia, where consumers will be only too happy to buy at the right price.
"(Iranian) oil will go somewhere else," Total SA Chief Executive Christophe de Margerie told Reuters in Davos on Wednesday. "Iran may give a discount to make it easier and quicker but nothing will change."
The net result is almost certain to be an overall increase in oil supply, initially into Asia but eventually to all world markets, and downward pressure on prices.
Expectations of improving supply are already beginning to affect prices, dampening Brent crude oil futures for nearby contracts relative to forward months, tipping the front of the price curve into a so-called contango.
The front-month contract for Brent, now March, traded around $111 per barrel on Thursday, and this week it has traded at a small discount to April. Many traders expect the rest of the price curve to move into contango as supplies improve.
The world's top oil exporter, Saudi Arabia, is pumping just under 10 million bpd and is most likely to make up any shortfall in Iranian supplies. It has promised to meet any extra requests from customers and Gulf industry sources expect a significant increase this summer, maybe of up to 500,000 bpd.
Iraq is aiming to expand its crude oil exports by up to 400,000 bpd by March, after starting up a new Gulf oil terminal this month. This would take its overall oil sales to about 2.5 million bpd, an Iraqi industry source says.
Libya, returning to full production after the overthrow of Muammar Gaddafi and civil war last year, has already pushed up oil exports to around 800,000 bpd this month, says the National Oil Corp. Libya expects to increase exports by up to 500,000 bpd by the third quarter.
"FAVOURABLE" PAYMENT TERMS
Meanwhile, many buyers of Iranian oil, especially in Asia, show no sign of supporting the Western campaign against Iran.
Chinese oil companies are negotiating hard with the state-run National Iranian Oil Company on term purchases and, while they want the lowest possible price, they have no intention of taking less Iranian crude, sources familiar with the Chinese term negotiations say.
India wants to take as much Iranian oil as it can because terms are "favorable," Oil Minister S. Jaipal Reddy said on Monday, after talks between the two sides on payment terms.
Together, China and India, could take more Iranian crude, possibly much more, if it were heavily discounted.
"Totting it all up, the figures show supplies from the MENA region improving, not decreasing," said a senior oil trader at a large U.S. bank. "We can't see a shortage coming."
David Wech, head of research at Vienna-based consultancy JBC Energy, said the price impact of the changing supply-demand picture had been obscured by worries over geopolitical risk:
"There is some logic that the situation might lead to ... more barrels if Iran manages to supply more than expected."
The big losers could be European refiners, already under huge pressure from poor margins and high debt. [ID:nL5E8CO097]
"The Iranians might have to discount a bit, but the real victims will be European refiners who will have to pay up for alternative supplies," said a senior trader at a large European refiner that has been a regular buyer of Iranian crude.
"The European refining sector is dying and the EU sanctions are another nail in the coffin."
(Editing by James Jukwey)
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Oscar nominations announced for supporting actor (AP)
BEVERLY HILLS, Calif. ? The 84th annual Academy Award nominations for supporting actor in a motion picture have been announced in Beverly Hills, Calif., by the Academy of Motion Picture Arts and Sciences.
The nominees announced Tuesday morning are: Kenneth Branagh, "My Week with Marilyn"; Jonah Hill, "Moneyball"; Nick Nolte, "Warrior"; Christopher Plummer, "Beginners"; Max von Sydow, "Extremely Loud & Incredibly Close."
The Oscars will be presented Feb. 26 at the Kodak Theatre in Los Angeles, hosted by Billy Crystal and broadcast live on ABC.
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Wednesday, January 25, 2012
The nations weather (AP)
Weather Underground Forecast for Tuesday, January 24, 2012.
Snowy conditions persist over the Southwest, scattered showers and thunderstorms develop along the Gulf Coast, and snow showers linger over the Great Lakes on Tuesday. A low pressure system that moved over California and into the Great Basin and Four Corners will continue moving southeastward over Colorado and New Mexico. This will allow for snow showers to diminish over the Great Basin and Arizona, while a few lingering snow showers will persist over Colorado and New Mexico. Snowfall accumulation in these areas will range from 3 to 5 inches. Meanwhile, as this system moves into the Southern Plains, it will pull additional moisture and energy onshore from the Gulf of Mexico. This will allow for scattered showers and thunderstorms to develop from the US and Mexico border, through Oklahoma, Louisiana, and Arkansas. Some of these storms will turn severe with strong winds, hail, and possibly a tornado or two. Expect rainfall totals to range from 1 to 2 inches, more likely in areas of severe thunderstorm development.
In the Northeast, a low pressure system that brought wintry weather to the Northeast and Great Lakes will advance into eastern Canada. However, the back side of this system will kick up some lake effect snow showers along the downwind shores of the Great Lakes. Snowfall accumulation will range from 2 to 3 inches in most of these areas. Elsewhere, precipitation across the East Coast will wind down as a cold front pushes offshore early Tuesday morning.
In the Northwest, another strong trough of low pressure moves onshore, producing another round of rain and high elevation snow for Washington, Oregon, and northern California. New snowfall totals will range from 4 to 6 inches in the Cascades, with 1 to 3 inches likely in far northern California. Temperatures in the Lower 48 states Monday have ranged from a morning low of -5 degrees at Gunnison, Colo. to a high of 87 degrees at Crystal River, Fla.
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Monday, January 9, 2012
Utah man pleads guilty to ex-girlfriend's murder (AP)
SALT LAKE CITY ? A Utah man was sentenced to 15 years to life in prison after pleading guilty to murdering his ex-girlfriend and hauling her dead body to Missouri inside a 50-gallon storage bin.
Michael Doyel entered the plea Friday and was immediately sentenced by Judge Judith Atherton in Salt Lake City's 3rd District Court.
In a parallel case, the 51-year-old Doyel pleaded guilty in December to a federal kidnapping charge. He was sentenced to 30 years in federal prison, and will serve that term before the Utah sentence.
In federal court, Doyel said he strangled Deborah Jones in April 2008 after she ended their relationship.
Jones' body was found inside a bin in the backseat of her car outside a Branson, Mo., motel.
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Saturday, January 7, 2012
Monday, January 2, 2012
19 Jan - 12 Apr, 2012: University of Georgia (GA)
01/19/2012
06/01/2012
05/31/2012
06/01/2012
05/31/2012
05/18/2012
05/31/2012
05/31/2012
05/25/2012
04/16/2012
01/12/2012
Downtown
05/02/2012
04/18/2012
04/12/2012
Downtown
04/19/2012
05/11/2012
01/27/2012
01/26/2012
04/24/2012
02/02/2012
02/29/2012
02/02/2012
02/02/2012
Capitol Hill
02/09/2012
& pizza dinner
02/15/2012
02/17/2012
02/15/2012
02/16/2012
Source: http://www.peacecorps.gov/index.cfm?shell=meet.regrec.event&eventid=99718&cid=rssatlanta
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